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A B6-deficient person starting semaglutide faces a double problem: their neurotransmitter synthesis is already compromised, and semaglutide further disrupts the GI and central nervous system pathways that depend on adequate B6 levels

Protein Research

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We also found that intra-arterial injection of anti-miR-96-5p into mice using microbubbles and an ultrasound system decreased GTRAP3-18 levels, leading to increased EAAC1 and GSH levels in the hippocampus [124]